Doria, A

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  • Doria, A (1)
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Author's Bibliography

Interactions between infections and immune-inflammatory cells in type 1 diabetes mellitus and inflammatory bowel diseases: evidences from animal models

Claesson, MH; Nicoletti, Ferdinando; Stošić-Grujičić, Stanislava; Doria, A; Zampieri, S

(2008)

TY  - JOUR
AU  - Claesson, MH
AU  - Nicoletti, Ferdinando
AU  - Stošić-Grujičić, Stanislava
AU  - Doria, A
AU  - Zampieri, S
PY  - 2008
UR  - https://radar.ibiss.bg.ac.rs/handle/123456789/1550
AB  - Type 1 diabetes mellitus (T1D) and inflammmtory bowel diseases (IBD) are multifactorial disorders of autoimmune origin. Several microbial agents have been reported to be associated with the development of type 1 diabetes and inflammatory bowel diseases in animal models by different mechanisms. These models which resemble the phenotype of the human disease they mimic, can be very useful to identify important pathogenetic mechanisms, as well as therapeutical targets to treat the disease. This review is focused on the immune inflammatory pathways which are considered to be associated with the pathogenesis T1D and IBD in transgenic mice.
T2  - Clinical and Experimental Rheumatology
T1  - Interactions between infections and immune-inflammatory cells in type 1 diabetes mellitus and inflammatory bowel diseases: evidences from animal models
IS  - 1
VL  - 26
EP  - S11
UR  - https://hdl.handle.net/21.15107/rcub_ibiss_1550
ER  - 
@article{
author = "Claesson, MH and Nicoletti, Ferdinando and Stošić-Grujičić, Stanislava and Doria, A and Zampieri, S",
year = "2008",
abstract = "Type 1 diabetes mellitus (T1D) and inflammmtory bowel diseases (IBD) are multifactorial disorders of autoimmune origin. Several microbial agents have been reported to be associated with the development of type 1 diabetes and inflammatory bowel diseases in animal models by different mechanisms. These models which resemble the phenotype of the human disease they mimic, can be very useful to identify important pathogenetic mechanisms, as well as therapeutical targets to treat the disease. This review is focused on the immune inflammatory pathways which are considered to be associated with the pathogenesis T1D and IBD in transgenic mice.",
journal = "Clinical and Experimental Rheumatology",
title = "Interactions between infections and immune-inflammatory cells in type 1 diabetes mellitus and inflammatory bowel diseases: evidences from animal models",
number = "1",
volume = "26",
pages = "S11",
url = "https://hdl.handle.net/21.15107/rcub_ibiss_1550"
}
Claesson, M., Nicoletti, F., Stošić-Grujičić, S., Doria, A.,& Zampieri, S.. (2008). Interactions between infections and immune-inflammatory cells in type 1 diabetes mellitus and inflammatory bowel diseases: evidences from animal models. in Clinical and Experimental Rheumatology, 26(1).
https://hdl.handle.net/21.15107/rcub_ibiss_1550
Claesson M, Nicoletti F, Stošić-Grujičić S, Doria A, Zampieri S. Interactions between infections and immune-inflammatory cells in type 1 diabetes mellitus and inflammatory bowel diseases: evidences from animal models. in Clinical and Experimental Rheumatology. 2008;26(1):null-S11.
https://hdl.handle.net/21.15107/rcub_ibiss_1550 .
Claesson, MH, Nicoletti, Ferdinando, Stošić-Grujičić, Stanislava, Doria, A, Zampieri, S, "Interactions between infections and immune-inflammatory cells in type 1 diabetes mellitus and inflammatory bowel diseases: evidences from animal models" in Clinical and Experimental Rheumatology, 26, no. 1 (2008),
https://hdl.handle.net/21.15107/rcub_ibiss_1550 .