TY  - JOUR
AU  - Daiber, Andreas
AU  - Kröller-Schön, Swenja
AU  - Frenis, Katie
AU  - Oelze, Matthias
AU  - Kalinović, Sanela
AU  - Vujačić-Mirski, Ksenija
AU  - Kuntić, Marin
AU  - Bayo Jimenez, Maria Teresa
AU  - Helmstädter, Johanna
AU  - Steven, Sebastian
AU  - Korać, Bato
AU  - Münzel, Thomas
PY  - 2019
UR  - https://onlinelibrary.wiley.com/doi/abs/10.1002/biof.1506
UR  - https://radar.ibiss.bg.ac.rs/handle/123456789/3329
AB  - Environmental noise is a well-recognized health risk and part of the external exposome-the World Health Organization estimates that 1 million healthy life years are lost annually in Western Europe alone due to noise-related complications, including increased incidence of hypertension, heart failure, myocardial infarction, and stroke. Previous data suggest that noise works through two paired pathways in a proposed reaction model for noise exposure. As a nonspecific stressor, chronic low-level noise exposure can cause a disruption of sleep and communication leading to annoyance and subsequent sympathetic and endocrine stress responses leading to increased blood pressure, heart rate, stress hormone levels, and in particular more oxidative stress, being responsible for vascular dysfunction and representing changes of the internal exposome. Chronic stress generates cardiovascular risk factors on its own such as increased blood pressure, blood viscosity, blood glucose, and activation of blood coagulation. To this end, persistent chronic noise exposure increases cardiometabolic diseases, including arterial hypertension, coronary artery disease, arrhythmia, heart failure, diabetes mellitus type 2, and stroke. The present review discusses the mechanisms of the nonauditory noise-induced cardiovascular and metabolic consequences, focusing on mental stress signaling pathways, activation of the hypothalamic-pituitary-adrenocortical axis and sympathetic nervous system, the association of these activations with inflammation, and the subsequent onset of oxidative stress and vascular dysfunction. © 2019 BioFactors, 2019.
T2  - BioFactors (Oxford, England)
T1  - Environmental noise induces the release of stress hormones and inflammatory signaling molecules leading to oxidative stress and vascular dysfunction-Signatures of the internal exposome.
DO  - 10.1002/biof.1506
ER  - 

@article{
author = "Daiber, Andreas and Kröller-Schön, Swenja and Frenis, Katie and Oelze, Matthias and Kalinović, Sanela and Vujačić-Mirski, Ksenija and Kuntić, Marin and Bayo Jimenez, Maria Teresa and Helmstädter, Johanna and Steven, Sebastian and Korać, Bato and Münzel, Thomas",
year = "2019",
abstract = "Environmental noise is a well-recognized health risk and part of the external exposome-the World Health Organization estimates that 1 million healthy life years are lost annually in Western Europe alone due to noise-related complications, including increased incidence of hypertension, heart failure, myocardial infarction, and stroke. Previous data suggest that noise works through two paired pathways in a proposed reaction model for noise exposure. As a nonspecific stressor, chronic low-level noise exposure can cause a disruption of sleep and communication leading to annoyance and subsequent sympathetic and endocrine stress responses leading to increased blood pressure, heart rate, stress hormone levels, and in particular more oxidative stress, being responsible for vascular dysfunction and representing changes of the internal exposome. Chronic stress generates cardiovascular risk factors on its own such as increased blood pressure, blood viscosity, blood glucose, and activation of blood coagulation. To this end, persistent chronic noise exposure increases cardiometabolic diseases, including arterial hypertension, coronary artery disease, arrhythmia, heart failure, diabetes mellitus type 2, and stroke. The present review discusses the mechanisms of the nonauditory noise-induced cardiovascular and metabolic consequences, focusing on mental stress signaling pathways, activation of the hypothalamic-pituitary-adrenocortical axis and sympathetic nervous system, the association of these activations with inflammation, and the subsequent onset of oxidative stress and vascular dysfunction. © 2019 BioFactors, 2019.",
journal = "BioFactors (Oxford, England)",
title = "Environmental noise induces the release of stress hormones and inflammatory signaling molecules leading to oxidative stress and vascular dysfunction-Signatures of the internal exposome.",
doi = "10.1002/biof.1506"
}

Daiber, A., Kröller-Schön, S., Frenis, K., Oelze, M., Kalinović, S., Vujačić-Mirski, K., Kuntić, M., Bayo Jimenez, M. T., Helmstädter, J., Steven, S., Korać, B.,& Münzel, T.. (2019). Environmental noise induces the release of stress hormones and inflammatory signaling molecules leading to oxidative stress and vascular dysfunction-Signatures of the internal exposome.. in BioFactors (Oxford, England).
https://doi.org/10.1002/biof.1506

Daiber A, Kröller-Schön S, Frenis K, Oelze M, Kalinović S, Vujačić-Mirski K, Kuntić M, Bayo Jimenez MT, Helmstädter J, Steven S, Korać B, Münzel T. Environmental noise induces the release of stress hormones and inflammatory signaling molecules leading to oxidative stress and vascular dysfunction-Signatures of the internal exposome.. in BioFactors (Oxford, England). 2019;.
doi:10.1002/biof.1506 .

Daiber, Andreas, Kröller-Schön, Swenja, Frenis, Katie, Oelze, Matthias, Kalinović, Sanela, Vujačić-Mirski, Ksenija, Kuntić, Marin, Bayo Jimenez, Maria Teresa, Helmstädter, Johanna, Steven, Sebastian, Korać, Bato, Münzel, Thomas, "Environmental noise induces the release of stress hormones and inflammatory signaling molecules leading to oxidative stress and vascular dysfunction-Signatures of the internal exposome." in BioFactors (Oxford, England) (2019),
https://doi.org/10.1002/biof.1506 . .