TY  - JOUR
AU  - Culita, Daniela C.
AU  - Dyakova, Lora
AU  - Marinescu, Gabriela
AU  - Zhivkova, Tanya
AU  - Georgieva, Milena
AU  - Vasileva, Bela
AU  - Spasov, Rossen
AU  - Miloshev, George
AU  - Kalfin, Reni
AU  - Vidaković, Melita
AU  - Oprea, Ovidiu
AU  - Alexandrova, Radostina
PY  - 2021
UR  - https://farmaciajournal.com/issue-articles/synthesis-characterization-and-cytotoxicity-evaluation-of-niii-cuii-and-znii-complexes-with-deoxycholate-ligand/
UR  - https://radar.ibiss.bg.ac.rs/handle/123456789/4446
AB  - Three new complexes of Ni(II), Cu(II) and Zn(II) with deoxycholate anion, of the type M(DCA)2 · nH2O (M = Cu(II) n = 4, M = Ni(II) n = 3 and M = Zn(II) n = 2) have been synthesized and characterized on the basis of elemental analysis, FTIR and UV-Vis spectroscopy, thermal analysis, molar conductivity and magnetic measurements. The cytotoxicity of the synthesized complexes was tested against HT29 human colorectal cancer cells. Applied at a concentration range of 10-200 µg/mL, sodium deoxycholate and its metal complexes have been found to decrease viability and growth of cultured HT29 cells in a time-and concentration-dependent manner in short-term and long-term experiments. The ability of the complexes to induce pathological changes, genotoxicity and apoptosis in the treated cells has also been proved. In addition, the complexes demonstrated a cytotoxic effect in HT29-OxPt (resistant to oxaliplatin) cells, allowing to conclude that they are more pronounced cytotoxic agents as compared to the sodium deoxycholate alone.
PB  - Bucuresti: Romanian Society for Pharmaceutical Sciences
T2  - Farmacia
T1  - Synthesis, characterization and cytotoxicity evaluation of ni(Ii), cu(ii) and zn(ii) complexes with deoxycholate ligand
IS  - 3
VL  - 69
DO  - 10.31925/farmacia.2021.3.7
SP  - 446
EP  - 460
ER  - 

@article{
author = "Culita, Daniela C. and Dyakova, Lora and Marinescu, Gabriela and Zhivkova, Tanya and Georgieva, Milena and Vasileva, Bela and Spasov, Rossen and Miloshev, George and Kalfin, Reni and Vidaković, Melita and Oprea, Ovidiu and Alexandrova, Radostina",
year = "2021",
abstract = "Three new complexes of Ni(II), Cu(II) and Zn(II) with deoxycholate anion, of the type M(DCA)2 · nH2O (M = Cu(II) n = 4, M = Ni(II) n = 3 and M = Zn(II) n = 2) have been synthesized and characterized on the basis of elemental analysis, FTIR and UV-Vis spectroscopy, thermal analysis, molar conductivity and magnetic measurements. The cytotoxicity of the synthesized complexes was tested against HT29 human colorectal cancer cells. Applied at a concentration range of 10-200 µg/mL, sodium deoxycholate and its metal complexes have been found to decrease viability and growth of cultured HT29 cells in a time-and concentration-dependent manner in short-term and long-term experiments. The ability of the complexes to induce pathological changes, genotoxicity and apoptosis in the treated cells has also been proved. In addition, the complexes demonstrated a cytotoxic effect in HT29-OxPt (resistant to oxaliplatin) cells, allowing to conclude that they are more pronounced cytotoxic agents as compared to the sodium deoxycholate alone.",
publisher = "Bucuresti: Romanian Society for Pharmaceutical Sciences",
journal = "Farmacia",
title = "Synthesis, characterization and cytotoxicity evaluation of ni(Ii), cu(ii) and zn(ii) complexes with deoxycholate ligand",
number = "3",
volume = "69",
doi = "10.31925/farmacia.2021.3.7",
pages = "446-460"
}

Culita, D. C., Dyakova, L., Marinescu, G., Zhivkova, T., Georgieva, M., Vasileva, B., Spasov, R., Miloshev, G., Kalfin, R., Vidaković, M., Oprea, O.,& Alexandrova, R.. (2021). Synthesis, characterization and cytotoxicity evaluation of ni(Ii), cu(ii) and zn(ii) complexes with deoxycholate ligand. in Farmacia
Bucuresti: Romanian Society for Pharmaceutical Sciences., 69(3), 446-460.
https://doi.org/10.31925/farmacia.2021.3.7

Culita DC, Dyakova L, Marinescu G, Zhivkova T, Georgieva M, Vasileva B, Spasov R, Miloshev G, Kalfin R, Vidaković M, Oprea O, Alexandrova R. Synthesis, characterization and cytotoxicity evaluation of ni(Ii), cu(ii) and zn(ii) complexes with deoxycholate ligand. in Farmacia. 2021;69(3):446-460.
doi:10.31925/farmacia.2021.3.7 .

Culita, Daniela C., Dyakova, Lora, Marinescu, Gabriela, Zhivkova, Tanya, Georgieva, Milena, Vasileva, Bela, Spasov, Rossen, Miloshev, George, Kalfin, Reni, Vidaković, Melita, Oprea, Ovidiu, Alexandrova, Radostina, "Synthesis, characterization and cytotoxicity evaluation of ni(Ii), cu(ii) and zn(ii) complexes with deoxycholate ligand" in Farmacia, 69, no. 3 (2021):446-460,
https://doi.org/10.31925/farmacia.2021.3.7 . .

TY  - CONF
AU  - Tolić, Anja
AU  - Ninković, Niccoleta Aleksandra
AU  - Rajić, Jovana
AU  - Đorđević, Miloš
AU  - Đorđević, Marija
AU  - Uskoković, Aleksandra
AU  - Grdović, Nevena
AU  - Mihailović, Mirjana
AU  - Arambašić Jovanović, Jelena
AU  - Dinić, Svetlana
AU  - Okamoto, Akimitsu
AU  - Vidaković, Melita
PY  - 2018
UR  - http://radar.ibiss.bg.ac.rs/handle/123456789/5799
AB  - Objectives: This study aim to implement a novel method, methylation-specific fluorescence in situ hybridization (MeFISH), based on microscopic visualization of DNA methylation/hydroxymethylation status at specific DNA regions in individual nuclei after pancreatic cell treatment with different compounds that possess a pronounced DNA (de)methylation capacity.
Methods: The DNA (de)methylating properties of two selected compounds caffeine (Co) and azacitidine (A) were evaluated in a Rin-5F pancreatic beta-cell line. Rin-5F cells were spin down on microscopic slides and further processed for preparing HALOs (relaxed DNA with preserved contacts with non-soluble nuclear proteins). The fluorescent visualization was achieved using ICON probe that covers region of interest in the promoter of the CXCL12 gene and target C positioned on the +26 bp, osmium for methylated cytosine (5mC)-dependent crosslinking and Tyramide Signal Amplification Systems for enhanced fluorescent staining.
Results: In control and Rin-5F cells treated with Co we were able to detect clear, single fluorescent signal that correspond to 5mC positioned on the +26 bp within the promoter region of the CXCL12 gene using MeFISH. Confirmation for the in situ hybridization specificity was achieved by omitting the crosslinking reaction with osmium. We observed a clear difference between control and Co treated samples, indicating that Co acts as pronounced DNA methylating compound. Treatment of cells with A lead to the appearance of a specific signal in a limited number of HALO preparations confirming demethylating property of A.
Conclusions: The Co acts as a pronounced DNA methylating agent in contrast to A, which demethylates CXCL12 gene and subsequently promotes higher gene expression. Higher methylation of the CXCL12 gene after cell treatment with Co leads to suppression of the gene which was observed by RT-qPCR. The analysed C, positioned on the +26 bp, may represent one of the major sites whose methylation is important for the regulation of the CXCL12 gene expression in vivo.
PB  - Karger Publishers
C3  - Selected Abstracts from the 3rd European Summer School on Nutrigenomics; 2018 Jun 25-29; Jesi, italy
T1  - MeFISH: Fluorescent detection of Target Methylated Cytosines within the Genome
DO  - 10.1159/000490753
SP  - 16
ER  - 

@conference{
author = "Tolić, Anja and Ninković, Niccoleta Aleksandra and Rajić, Jovana and Đorđević, Miloš and Đorđević, Marija and Uskoković, Aleksandra and Grdović, Nevena and Mihailović, Mirjana and Arambašić Jovanović, Jelena and Dinić, Svetlana and Okamoto, Akimitsu and Vidaković, Melita",
year = "2018",
abstract = "Objectives: This study aim to implement a novel method, methylation-specific fluorescence in situ hybridization (MeFISH), based on microscopic visualization of DNA methylation/hydroxymethylation status at specific DNA regions in individual nuclei after pancreatic cell treatment with different compounds that possess a pronounced DNA (de)methylation capacity.
Methods: The DNA (de)methylating properties of two selected compounds caffeine (Co) and azacitidine (A) were evaluated in a Rin-5F pancreatic beta-cell line. Rin-5F cells were spin down on microscopic slides and further processed for preparing HALOs (relaxed DNA with preserved contacts with non-soluble nuclear proteins). The fluorescent visualization was achieved using ICON probe that covers region of interest in the promoter of the CXCL12 gene and target C positioned on the +26 bp, osmium for methylated cytosine (5mC)-dependent crosslinking and Tyramide Signal Amplification Systems for enhanced fluorescent staining.
Results: In control and Rin-5F cells treated with Co we were able to detect clear, single fluorescent signal that correspond to 5mC positioned on the +26 bp within the promoter region of the CXCL12 gene using MeFISH. Confirmation for the in situ hybridization specificity was achieved by omitting the crosslinking reaction with osmium. We observed a clear difference between control and Co treated samples, indicating that Co acts as pronounced DNA methylating compound. Treatment of cells with A lead to the appearance of a specific signal in a limited number of HALO preparations confirming demethylating property of A.
Conclusions: The Co acts as a pronounced DNA methylating agent in contrast to A, which demethylates CXCL12 gene and subsequently promotes higher gene expression. Higher methylation of the CXCL12 gene after cell treatment with Co leads to suppression of the gene which was observed by RT-qPCR. The analysed C, positioned on the +26 bp, may represent one of the major sites whose methylation is important for the regulation of the CXCL12 gene expression in vivo.",
publisher = "Karger Publishers",
journal = "Selected Abstracts from the 3rd European Summer School on Nutrigenomics; 2018 Jun 25-29; Jesi, italy",
title = "MeFISH: Fluorescent detection of Target Methylated Cytosines within the Genome",
doi = "10.1159/000490753",
pages = "16"
}

Tolić, A., Ninković, N. A., Rajić, J., Đorđević, M., Đorđević, M., Uskoković, A., Grdović, N., Mihailović, M., Arambašić Jovanović, J., Dinić, S., Okamoto, A.,& Vidaković, M.. (2018). MeFISH: Fluorescent detection of Target Methylated Cytosines within the Genome. in Selected Abstracts from the 3rd European Summer School on Nutrigenomics; 2018 Jun 25-29; Jesi, italy
Karger Publishers., 16.
https://doi.org/10.1159/000490753

Tolić A, Ninković NA, Rajić J, Đorđević M, Đorđević M, Uskoković A, Grdović N, Mihailović M, Arambašić Jovanović J, Dinić S, Okamoto A, Vidaković M. MeFISH: Fluorescent detection of Target Methylated Cytosines within the Genome. in Selected Abstracts from the 3rd European Summer School on Nutrigenomics; 2018 Jun 25-29; Jesi, italy. 2018;:16.
doi:10.1159/000490753 .

Tolić, Anja, Ninković, Niccoleta Aleksandra, Rajić, Jovana, Đorđević, Miloš, Đorđević, Marija, Uskoković, Aleksandra, Grdović, Nevena, Mihailović, Mirjana, Arambašić Jovanović, Jelena, Dinić, Svetlana, Okamoto, Akimitsu, Vidaković, Melita, "MeFISH: Fluorescent detection of Target Methylated Cytosines within the Genome" in Selected Abstracts from the 3rd European Summer School on Nutrigenomics; 2018 Jun 25-29; Jesi, italy (2018):16,
https://doi.org/10.1159/000490753 . .

TY  - CONF
AU  - Rajić, Jovana
AU  - Grdović, Nevena
AU  - Petrović Matić, Sanja
AU  - Dinić, Svetlana
AU  - Uskoković, Aleksandra
AU  - Mihailović, Mirjana
AU  - Arambašić Jovanović, Jelena
AU  - Tolić, Anja
AU  - Đorđević, Marija
AU  - Đorđević, Miloš
AU  - Poznanović, Goran
AU  - Pucar, Ana
AU  - Vidaković, Melita
PY  - 2018
UR  - http://radar.ibiss.bg.ac.rs/handle/123456789/5800
AB  - Objectives: Procalcitonin (PCT) has recently emerged as an important biomarker for an early and accurate diagnosis of bacte­rial infection, hence suggested as biomarker of periodontal disease caused by oral pathogenic microorganisms. Altered DNA meth­ylation of PCT coding gene - calcitonin-related polypeptide a (CALCA) has been shown during systemic inflammation. The aim of this study was to evaluate the influence oflocal and/or systemic inflammation present during periodontitis and diabetes on DNA methylation status of CALCA and its potential use as epigenetic­based biomarker of these chronic inflammatory conditions. 
Methods: The study included 65 individuals divided in three groups: healthy control (n = 17), periodontitis (n = 27) and diabe­tes/periodontitis group (n = 21). Periodontitis was diagnosed us­ing International Workshop for a Classification of Periodontal Diseases and Conditions criteria (1999) while type 2 diabetes as­sessment was performed according to WHO criteria (2013). DNA methylation profile of CALCA promoter in buccal epithelial cells was analyzed by methylation specific polymerase chain reaction (MSP). 
Results: Decrease in DNA methylation of CALCA promoter was observed in periodontitis and even more pronounced in dia­betes/periodontitis compared to control group, although without statistical significance. Correlation analysis revealed statistically significant relationship between the extent of DNA methylation of the CALCA promoter and glycosylated hemoglobin. Even though it is known that life style affects DNA methylation patterns, there was no difference in DNA methylation of CALCA promoter be­tween smokers/non-smokers and normal/overweight individuals. 
Conclusions: Presented results suggest that local periodontal inflammation contributes to the change, but that only systemic inflammation significantly alters the DNA methylation profile of CALCA in buccal cells. Those results imply that DNA methylation status of CALCA reflects systemic inflammation, but additional studies are needed to estimate usefulness of this epigenetic-based biomarker for periodontal disease.
PB  - Krager Publishers
C3  - Selected Abstracts from the 3rd European Summer School on Nutrigenomics; 2018 Jun 25-29; Jesi, italy
T1  - Evaluation of the DNA Methylation Status of Procalcitonin Gene as a Biomarker of Local and Systemic Inflammation
DO  - 10.1159/000490753
SP  - 12
ER  - 

@conference{
author = "Rajić, Jovana and Grdović, Nevena and Petrović Matić, Sanja and Dinić, Svetlana and Uskoković, Aleksandra and Mihailović, Mirjana and Arambašić Jovanović, Jelena and Tolić, Anja and Đorđević, Marija and Đorđević, Miloš and Poznanović, Goran and Pucar, Ana and Vidaković, Melita",
year = "2018",
abstract = "Objectives: Procalcitonin (PCT) has recently emerged as an important biomarker for an early and accurate diagnosis of bacte­rial infection, hence suggested as biomarker of periodontal disease caused by oral pathogenic microorganisms. Altered DNA meth­ylation of PCT coding gene - calcitonin-related polypeptide a (CALCA) has been shown during systemic inflammation. The aim of this study was to evaluate the influence oflocal and/or systemic inflammation present during periodontitis and diabetes on DNA methylation status of CALCA and its potential use as epigenetic­based biomarker of these chronic inflammatory conditions. 
Methods: The study included 65 individuals divided in three groups: healthy control (n = 17), periodontitis (n = 27) and diabe­tes/periodontitis group (n = 21). Periodontitis was diagnosed us­ing International Workshop for a Classification of Periodontal Diseases and Conditions criteria (1999) while type 2 diabetes as­sessment was performed according to WHO criteria (2013). DNA methylation profile of CALCA promoter in buccal epithelial cells was analyzed by methylation specific polymerase chain reaction (MSP). 
Results: Decrease in DNA methylation of CALCA promoter was observed in periodontitis and even more pronounced in dia­betes/periodontitis compared to control group, although without statistical significance. Correlation analysis revealed statistically significant relationship between the extent of DNA methylation of the CALCA promoter and glycosylated hemoglobin. Even though it is known that life style affects DNA methylation patterns, there was no difference in DNA methylation of CALCA promoter be­tween smokers/non-smokers and normal/overweight individuals. 
Conclusions: Presented results suggest that local periodontal inflammation contributes to the change, but that only systemic inflammation significantly alters the DNA methylation profile of CALCA in buccal cells. Those results imply that DNA methylation status of CALCA reflects systemic inflammation, but additional studies are needed to estimate usefulness of this epigenetic-based biomarker for periodontal disease.",
publisher = "Krager Publishers",
journal = "Selected Abstracts from the 3rd European Summer School on Nutrigenomics; 2018 Jun 25-29; Jesi, italy",
title = "Evaluation of the DNA Methylation Status of Procalcitonin Gene as a Biomarker of Local and Systemic Inflammation",
doi = "10.1159/000490753",
pages = "12"
}

Rajić, J., Grdović, N., Petrović Matić, S., Dinić, S., Uskoković, A., Mihailović, M., Arambašić Jovanović, J., Tolić, A., Đorđević, M., Đorđević, M., Poznanović, G., Pucar, A.,& Vidaković, M.. (2018). Evaluation of the DNA Methylation Status of Procalcitonin Gene as a Biomarker of Local and Systemic Inflammation. in Selected Abstracts from the 3rd European Summer School on Nutrigenomics; 2018 Jun 25-29; Jesi, italy
Krager Publishers., 12.
https://doi.org/10.1159/000490753

Rajić J, Grdović N, Petrović Matić S, Dinić S, Uskoković A, Mihailović M, Arambašić Jovanović J, Tolić A, Đorđević M, Đorđević M, Poznanović G, Pucar A, Vidaković M. Evaluation of the DNA Methylation Status of Procalcitonin Gene as a Biomarker of Local and Systemic Inflammation. in Selected Abstracts from the 3rd European Summer School on Nutrigenomics; 2018 Jun 25-29; Jesi, italy. 2018;:12.
doi:10.1159/000490753 .

Rajić, Jovana, Grdović, Nevena, Petrović Matić, Sanja, Dinić, Svetlana, Uskoković, Aleksandra, Mihailović, Mirjana, Arambašić Jovanović, Jelena, Tolić, Anja, Đorđević, Marija, Đorđević, Miloš, Poznanović, Goran, Pucar, Ana, Vidaković, Melita, "Evaluation of the DNA Methylation Status of Procalcitonin Gene as a Biomarker of Local and Systemic Inflammation" in Selected Abstracts from the 3rd European Summer School on Nutrigenomics; 2018 Jun 25-29; Jesi, italy (2018):12,
https://doi.org/10.1159/000490753 . .

TY  - CONF
AU  - Rajić, Jovana
AU  - Grdović, Nevena
AU  - Inić-Kanada, Aleksandra
AU  - Stein, Elisabath
AU  - Schuerer, Nadine
AU  - Uskoković, Aleksandra
AU  - Ghasemian, Ehsan
AU  - Dinić, Svetlana
AU  - Mihailović, Mirjana
AU  - Arambašić Jovanović, Jelena
AU  - Tolić, Anja
AU  - Đorđević, Marija
AU  - Đorđević, Miloš
AU  - Poznanović, Goran
AU  - Barisani-Asenbauer, Talin
AU  - Vidaković, Melita
PY  - 2017
UR  - http://radar.ibiss.bg.ac.rs/handle/123456789/5814
AB  - Repeated eye infections with Chlamydia trachomatis (Ct) usually lead to conjunctiva! scarring, corneal damage and blindness (trachoma). Considering epithelial to mesenchymal transition (EMD as unavoidable player in the development of fibrosis and taking into account that EMT is epigenetically reg­ulated process, our intention was to explore whether Ct infec­tion induces EMT in vitro and to uncover potential underlying epigenetic mechanisms. 
Human conjunctiva! epithelial cells were infected with 107 IFU of Ct for 72 h. The mRNA and protein expression of EMT mark­ers (E-cadherin and fibronectin) were assessed by RT-qPCR, lmmunoblotting and lmmunocytochemfstry. DNA methyfation analysis of selected regions of marker genes were examined by MSP, HRM and Bs-Seq. 
The Ct infection induced EMT-related changes in mRNA and protein expression, downregulation of epithelial marker E-cadherin and upregulation of mesenchymal marker fibronectin. Increase in DNA methylation of E-cadherin gene promoter was in correlation with its decreased expression, while DNA meth­ylation status of fibronectin gene couldn't be related to its ex­pression level. 
We suggest that manipulating the EMT process via modula­tion of DNA methylation of E-cadherin opens up possibilities to stop or revers the progression of scarring as a new strategy in trachoma treatment.
PB  - Institute of Biology and Immunology of Reproduction, BAS, COST Action CA16119 and Mouseprint Ltd.
C3  - Proceedings of CellFit meeting: In vitro 3-D total cell guidance and fitness; 2017 Sep 12-14; Albena Resort, Bulgaria
T1  - Chlamydia trachomatis infection triggers  Epithelial Mesenchymal Transition in  conjunctiva: involvement of DNA methylation
SP  - 64
EP  - 65
UR  - https://hdl.handle.net/21.15107/rcub_ibiss_5814
ER  - 

@conference{
author = "Rajić, Jovana and Grdović, Nevena and Inić-Kanada, Aleksandra and Stein, Elisabath and Schuerer, Nadine and Uskoković, Aleksandra and Ghasemian, Ehsan and Dinić, Svetlana and Mihailović, Mirjana and Arambašić Jovanović, Jelena and Tolić, Anja and Đorđević, Marija and Đorđević, Miloš and Poznanović, Goran and Barisani-Asenbauer, Talin and Vidaković, Melita",
year = "2017",
abstract = "Repeated eye infections with Chlamydia trachomatis (Ct) usually lead to conjunctiva! scarring, corneal damage and blindness (trachoma). Considering epithelial to mesenchymal transition (EMD as unavoidable player in the development of fibrosis and taking into account that EMT is epigenetically reg­ulated process, our intention was to explore whether Ct infec­tion induces EMT in vitro and to uncover potential underlying epigenetic mechanisms. 
Human conjunctiva! epithelial cells were infected with 107 IFU of Ct for 72 h. The mRNA and protein expression of EMT mark­ers (E-cadherin and fibronectin) were assessed by RT-qPCR, lmmunoblotting and lmmunocytochemfstry. DNA methyfation analysis of selected regions of marker genes were examined by MSP, HRM and Bs-Seq. 
The Ct infection induced EMT-related changes in mRNA and protein expression, downregulation of epithelial marker E-cadherin and upregulation of mesenchymal marker fibronectin. Increase in DNA methylation of E-cadherin gene promoter was in correlation with its decreased expression, while DNA meth­ylation status of fibronectin gene couldn't be related to its ex­pression level. 
We suggest that manipulating the EMT process via modula­tion of DNA methylation of E-cadherin opens up possibilities to stop or revers the progression of scarring as a new strategy in trachoma treatment.",
publisher = "Institute of Biology and Immunology of Reproduction, BAS, COST Action CA16119 and Mouseprint Ltd.",
journal = "Proceedings of CellFit meeting: In vitro 3-D total cell guidance and fitness; 2017 Sep 12-14; Albena Resort, Bulgaria",
title = "Chlamydia trachomatis infection triggers  Epithelial Mesenchymal Transition in  conjunctiva: involvement of DNA methylation",
pages = "64-65",
url = "https://hdl.handle.net/21.15107/rcub_ibiss_5814"
}

Rajić, J., Grdović, N., Inić-Kanada, A., Stein, E., Schuerer, N., Uskoković, A., Ghasemian, E., Dinić, S., Mihailović, M., Arambašić Jovanović, J., Tolić, A., Đorđević, M., Đorđević, M., Poznanović, G., Barisani-Asenbauer, T.,& Vidaković, M.. (2017). Chlamydia trachomatis infection triggers  Epithelial Mesenchymal Transition in  conjunctiva: involvement of DNA methylation. in Proceedings of CellFit meeting: In vitro 3-D total cell guidance and fitness; 2017 Sep 12-14; Albena Resort, Bulgaria
Institute of Biology and Immunology of Reproduction, BAS, COST Action CA16119 and Mouseprint Ltd.., 64-65.
https://hdl.handle.net/21.15107/rcub_ibiss_5814

Rajić J, Grdović N, Inić-Kanada A, Stein E, Schuerer N, Uskoković A, Ghasemian E, Dinić S, Mihailović M, Arambašić Jovanović J, Tolić A, Đorđević M, Đorđević M, Poznanović G, Barisani-Asenbauer T, Vidaković M. Chlamydia trachomatis infection triggers  Epithelial Mesenchymal Transition in  conjunctiva: involvement of DNA methylation. in Proceedings of CellFit meeting: In vitro 3-D total cell guidance and fitness; 2017 Sep 12-14; Albena Resort, Bulgaria. 2017;:64-65.
https://hdl.handle.net/21.15107/rcub_ibiss_5814 .

Rajić, Jovana, Grdović, Nevena, Inić-Kanada, Aleksandra, Stein, Elisabath, Schuerer, Nadine, Uskoković, Aleksandra, Ghasemian, Ehsan, Dinić, Svetlana, Mihailović, Mirjana, Arambašić Jovanović, Jelena, Tolić, Anja, Đorđević, Marija, Đorđević, Miloš, Poznanović, Goran, Barisani-Asenbauer, Talin, Vidaković, Melita, "Chlamydia trachomatis infection triggers  Epithelial Mesenchymal Transition in  conjunctiva: involvement of DNA methylation" in Proceedings of CellFit meeting: In vitro 3-D total cell guidance and fitness; 2017 Sep 12-14; Albena Resort, Bulgaria (2017):64-65,
https://hdl.handle.net/21.15107/rcub_ibiss_5814 .